Trenbolone Enanthate is a 19-nortestosterone (19-nor) anabolic androgenic steroid. The 19-nor label refers to a structural change of the testosterone hormone in that it lacks a carbon atom at the 19th position. The Trenbolone hormone is the Nandrolone hormone with an added double bond at carbons 9 and 11. This structural change slows its metabolism, greatly increases its binding affinity to the androgen receptor and inhibits it from aromatizing. While this is truly a slight modification, it truly creates a different hormone and one that is far more powerful than Nandrolone.
Apigenin acts as a monoamine transporter activator, one of the few chemicals demonstrated to possess this property.  Apigenin is a ligand for central benzodiazepine receptors that competitively inhibited the binding of flunitrazepam with a K i of 4 μM, exerting anxiolytic and slight sedative effects.  Apigenin shows second-order positive modulatory activity at GABA A receptors.   It has also effects on adenosine receptors  and is an acute antagonist at the NMDA receptors ( IC 50 = 10 μM).  In addition, like various other flavonoids, apigenin has been found to possess nanomolar affinity for the opioid receptors (K e = 410 nM, 970 nM, and 410 nM for the μ- , δ- , and κ-opioid receptors , respectively), acting as a non-selective antagonist of all three opioid receptors. 
DHEA is transformed into DHEA-S by sulfation at the C3β position via the sulfotransferase enzymes SULT2A1 and to a lesser extent SULT1E1 .    This occurs naturally in the adrenal cortex and during first-pass metabolism in the liver and intestines when exogenous DHEA is administered orally. [ citation needed ] Levels of DHEA-S in circulation are approximately 250 to 300 times those of DHEA.  DHEA-S in turn can be converted back into DHEA in peripheral tissues via steroid sulfatase (STS).